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Lessons from animal diabetes II. John Libbey, London Paris, pp 112–116Google Scholar. Wang Y, Hao L, Gill RG, Lafferty KJ (1987) Autoimmune diabetes in NOD mouse is L3T4 T-lymphocyte dependent. Diabetes 36: 535–538Google Scholar. 6. Bendelac A, Carnaud C, Boitard C, Bach JF (1987) Syngeneic transfer of autoimmune diabetes from diabetic NOD mice to healthy neonates. Requirement for both L3T4+ and Lyt-2 T cells. J Exp Med 166: 823–832Google Scholar. 7. Offner . Thieme T, Vandenbark AA (1987) Gangliosides induce selective modulation of CD4 from helper T lymphocytes.
The book discusses nutritionally diabetes-prone animals and considerations of insulin resistance and obesity. The contributors also address the importance of recent findings on the pathogenesis of diabetes and its complications in relation to human disease. Including contributions from prominent experts in the field, the book brings together scattered data and lucidly presents it. This promotes the understanding of the etiopathology of diabetes and offers a new grasp of the insulin action, its negative feedback leading to insulin resistance, and its detrimental outcomes. 271. Nutritionally Induced Insulin Resistance Diabetes and Beta Cell Loss.
PDF On Jan 1, 2001, Yasunori Kanazawa and others published Lessons from Animal Diabetes 8, 24–26 July 2001. 247 · Source: PubMed Central. Cite this publication.
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It is also a rich and useful reference source for graduate students in these fields
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Diabetes in the Chinese hamster. 267. Studies on the pathogenesis of NIDDM in the.
Five articles are devoted to this topic ranging from the B-cell function in transgenic animals to the various effects on diabetes complications. The section on NIDDM, comprising of 10 articles, deals both with new and existing models, their particular widely varying pathogenesis, genetic character istics and complications. Diabetes in the Chinese hamster. 307. Antidiabetic characteristics of ne in animal. 349. Endoneurial vessel abnormalities in diabetic animal models.
The Zucker diabetic fatty rat: lessons from leptin receptor deficiency.
Autoimmune diabetes, present status of knowledge in animals and the implications for the understanding and preventing of human type 1 diabetes. Diabesity in ob/ob and db/db mice: the use of these models for studies of diabetes and obesity in humans. The Zucker diabetic fatty rat: lessons from leptin receptor deficiency. The Goto-Kaizaki diabetic rat evolved by selective inbreeding. Then NSY mouse, an animal model with human type 2 diabetes with polygenic inheritance.
Frontiers in Endocrinology. Diabetes development was then monitored to determine if B cells expressing separate subcongenic intervals from the NOR strain protected recipient mice from T1D compared to standard NOD B cells. Toggle navigation Section. The results established that at least four adjacent intervals interactively contribute to how diabetogenic B cells become tolerized or cause T1D, including processes that increase the efficiency of B cell anergy or B cell hyperresponsiveness to B cell receptor stimulation.